By Andrew Krentz
Chapter 1 Pathophysiology of Insulin Resistance (pages 1–49):
Chapter 2 Insulin Resistance in scientific medication (pages 50–118):
Chapter three administration of Insulin Resistance and linked stipulations (pages 119–177):
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Additional resources for Insulin Resistance: A Clinical Handbook
J. (2001) A model-based method for assessing insulin sensitivity from the oral glucose tolerance test. Diabetes Care24,539-548. Mauriege, P. & Bouchard, C. (1996) Trp64Arg mutation in P,-adrenoceptor gene of d o u b t h l significance for obesity and insulin resistance. Lancet 348,698-699. A. D. (1996) Hexosamines and insulin resistance. Diabetes 45, 1003-1 009. Moller, D. ) (1993) Insulin Resistance, John Wiley, Chichester. E. S. (1991) Insulin resistance-mechanisms, 48 PATHOPHYSIOLOGY OF INSULIN RESISTANCE syndromes, and implications.
1). Much more common is the wellrecognized association between maternal third trimester hyperglycaemia and macrosomia. This is held to be a consequence of the growth-promoting consequences of fetal hyperinsulinaemia which in turn results from P-cell stimulation by increased transfer of nutrients across the placenta. In the Pima Indians of Arizona, who have the highest recorded prevalence of type 2 diabetes, the risk of adult diabetes is related both to low and high birth weight in a bimodal relationship.
1 Clinical features Common inherited or ncqztired disorders The clinical relevance of insulin resistance is uncertain in inany conditions Insulin resistance per se is asymptomatic 50 Many common inherited or acquired disorders in humans are associated with insulin resistance. g. impaired glucose tolerance or type 2 diabetes, the insulin resistance is almost universal and is regarded as a major factor in the aetiology of the disorder. g. polycystic ovary disease, insulin resistance is emerging as an important target for therapeutic intervention.
Insulin Resistance: A Clinical Handbook by Andrew Krentz