New PDF release: The Encyclopedia Of Molecular Biology

By Thomas E. Creighton

ISBN-10: 0471153028

ISBN-13: 9780471153023

The sector of molecular biology has revolutionized the examine of biology. The purposes to drugs are huge, immense, starting from diagnostic options for disorder and genetic issues, to medications, to gene remedy. targeting the basics of molecular biology and encompassing all points of the expression of genetic info, the Encyclopedia of Molecular Biology turns into the 1st element of reference for either newbies and verified pros in molecular biology wanting to profit approximately any specific element of the sphere.

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Extra info for The Encyclopedia Of Molecular Biology

Sample text

They are formed as a result of chromosomal damage. Lacking a centromere, there is no means of attachment to the mitotic spindle, which leads to a failure to segregate replicated chromatids in the acentric fragments to the daughter cells during cell division. Therefore, the acentric fragments are normally lost from most cells with progressive cell cycles. The breakage of the chromosome that separates the acentric fragment from the remainder of the chromosome containing the centromere can be a result of either inherent chromosomal fragility, radiation damage, or defective DNA repair mechanisms.

Specific aminoacylation of tRNA by their cognate aminoacyl-tRNA synthetases is dependent on the presence of a series of identity elements. Limited in number, these elements are preferentially located in the anticodon loop and in the acceptor stem (2-4). Residue 73, next to the CCA end, is called the “discriminator” base. The hypothesis that it contributes to discrimination of tRNA by cognate aminoacyl tRNA synthetases (5) has been largely confirmed. Residue 73 contributes strongly to specific aminoacylation of 17 different Escherichia coli tRNAs (6).

A fragile site associated with a form of inherited mental retardation known as fragile X mental retardation has been characterized in some detail (5). A gene called FMR-1 (fragile X mental retardation gene 1) is present in the fragile site. This gene contains within it a trinucleotide repeat of CGG. A normal individual has between 6 and 50 of these trinucleotide repeats. Individuals with fragile X syndrome contain expansions of these repeat sequences such that they have several hundred CGG triplets.

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The Encyclopedia Of Molecular Biology by Thomas E. Creighton


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